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These findings suggest that suppressing Nrf2 signalling may be critical for the ECM remodelling produced by mechanical damage (Li et al
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As it would not have been possible to distinguish the effects of changes in DTC-AC levels from those of a reduction in lag-2 expression, we did not measure GLP-1 activity in daf-7 mutants
Elucidating these interactions may ultimately refine precision approaches to incretin-based therapy in metabolic disease
The Follow-Up: Ongoing Support
Mechanism involving anti-apoptotic signaling and trophic support
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