doi:10.1016/j.ecoenv.2022.114057 251
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World J Gastroenterol (2013) 19(46):8489501
doi: 10.1080/01443615.2019.1604644 146 SalehiBBerkay YlmazYAntikaGBoyunegmez TumerTFawzi MahomoodallyMLobineDet al
This apparent paradox, largely attributed to context-dependent mechanisms, has remained incompletely understood
Under TNF-induced inflammatory conditions, GLP-1RAs (such as Exendin-4 and GLP-1) suppress the production of MMPs in human coronary artery smooth muscle cells (hCASMCs) by inhibiting AKT-Thr308 phosphorylation