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This study demonstrated the importance of combined GLP-1R and GCGR signalling, as administration of the balanced co-agonist to Glp1r -/- mice led to hyperglycaemia, further demonstrating the necessity of GLP-1R and GCGR co-agonism to minimise this predicted side-effect

PMID: 37250165

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Inflammopharmacology 25(1):1124 von Blow V, Dubben S, Engelhardt G, Hebel S, Plmkers B, Heine H, Rink L, Haase H (2017) Zinc-dependent suppression of TNF-alpha production is mediated by protein kinase A-induced inhibition of Raf-1, I kappa B kinase beta, and NF-kappa B

Clinical and Experimental Pharmacology and Physiology, 47 , 927939

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