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In the aging heart that has been pretreated with acetylcarnitine and exposed to ischemia-reperfusion, the mitochondrial defect due to aging is eliminated, and only the defect due to ischemia is still present, improving the recovery after ischemia-reperfusion to the level of the adult heart [28]

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Always check for visual changes before reconstituting a stored vial

To answer the first question, we transplanted tumors lacking the ability to produce GSH (Gclc-KO) into wild-type mice and, surprisingly, the tumors grew normally

The peptide sequence (glycine-histidine-lysine) chelates copper ions in a way that allows controlled cellular uptake without the oxidative damage that free copper can cause

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