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[DOI] [PubMed] [Google Scholar] Wang Z, Sun J, Zu X, et al

The treated mice also had a higher ratio of glutathione to glutathione disulfide, the molecule that glutathione becomes after it neutralizes free radicals

All evidence is from animal models, with no published human data

Thus, our study provides a new candidate molecule for drug research in treating AD and illustrates that intracerebral GLP-1R activation, which facilitates astrocyte-neuron metabolic coupling, may be a potential approach for the treatment of AD

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Incretin mimetics delay gastric emptying, enhance satiety, and reduce appetite through central nervous system effects, leading to reduced caloric intake and 26 kg weight loss on average