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During development and progression of atherosclerosis, Nrf2 signaling modulates many physiological and pathophysiological processes, like regulation of lipid homeostasis, CD36 gene expression regulation, foam cell formation, macrophage polarization, immunity regulation (Th2 differentiation and inhibition of pro-inflammatory gene expression through NFB down-regulation), redox regulation and inflammation, improvement of endothelial dysfunction, as well as GSH synthesis and utilization (234245)

Maintaining balanced meals, good hydration, regular sleep, physical activity, and ongoing clinical support may help reduce stress, fatigue, and mood related symptoms over time

Model performance was evaluated using cross-validated area under the receiver operating characteristic curve (AUROC)

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Instead, treatment is categorized into behavioral/non-pharmacologic, oral medicines, bladder instillations, procedures, and major surgery