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Furthermore, CRIP2 depletion and copper-induced CRIP2 degradation increase reactive oxygen species (ROS) levels and activate autophagy in H1299 cells

The study was supported by a National Institutes of Health (NIH) grant HD084422 to D.M.-R

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Posttranslational modifications (PTMs), including phosphorylation, acetylation, methylation, and ubiquitination, play key roles in the regulation of p53