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Loss of farnesoid X receptor (FXR) exacerbated the ferroptosis signaling pathway by enhancing iron accumulation, increasing lipid peroxidation, decreasing GSH levels and reducing GPX4 expression in cisplatin-treated mice and HK-2 cells [175]

Results in C3H mice

Ramirez Bosca A, Carrion Gutierrez MA, Soler A, et al

Both PENDRIN and NIS were expressed on the surface of human leukocytes

This results in activation of two major canonical signaling pathways: the IRS-1/PI3K/Akt pathway, which is linked to most metabolic actions, such as stimulation of glucose uptake, lipogenesis, glycogen synthesis, and inhibition of gluconeogenesis, and the Shc/Grb2/Sos/Ras/Raf/MAPK pathway, which regulates cell growth and differentiation (5, 7)

Furthermore, periosteum bone formation rate and mineralizing surface also appeared to be preserved and several-fold higher than in catabolic agent-only models, partially restoring the suppressed bone formation