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Like GIPR agonism 14 , we find that the body weight and food intake reducing effects of GIPR antagonism are lost in global Gipr -deficient mice

Mice fed a high-fat diet are able to lower their blood glucose levels when L-carnitine supplementation is given to them, as opposed to mice fed a high-fat diet without the supplement (3)

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Methylation pathways become saturated, and excess methionine is converted to homocysteine, which may contribute to cardiovascular risk if B-vitamin cofactors (B6, B12, folate) aren't adequate