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"Hepatocyte-specific Gclc deletion leads to rapid onset of steatosis with mitochondrial injury and liver failure"

GHK-Cu is one of the most extensively characterized signaling peptides in human biology, modulating over 4,000 genes including upregulation of tissue repair, collagen synthesis, and antioxidant defense pathways, while downregulating inflammatory and cellular senescence-associated genes

FAK is overexpressed in a large number of human cancers (5080% of all solid tumors 1 ) and modulates multiple hallmarks of cancer, including invasion, metastasis, resistance to apoptosis, proliferation, glycolysis, angiogenesis, and immune inactivation 2,3,4,5

GHK-Cu acts on surface regeneration (collagen, elastin, gene expression, skin anti-aging)

Rollini M, Musatti A, Manzoni M (2010) Production of glutathione in extracellular form by Saccharomyces cerevisiae

Decreased lipogenesis: The process of generating new fat