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In particular, we found that the moderate depletion of GSH is operative upon fasting conditions in several organs including skeletal muscle and brain and this event is causative of the augmentation of NO availability

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Additionally, it suppresses pro-inflammatory cytokines and chemokines via the NF-B pathway while reducing reactive oxygen species (ROS) production [51]

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Concurrently, their antioxidant, anti-inflammatory, and neurotrophic actions help protect neuronal integrity from stress-induced damage, collectively supporting recovery of thyroidbrain axis function and neurocognitive performance (184, 194)