-hydroxybutyric acid (-HB), which was lower in PD patients, may reflect changes in glutathione metabolism and the oxidative stress response
Available from: De Stefano ME, Herrero MT
With the current absence of technologies to rectify the actual mutations that underlie mitochondrial diseases, medical science can only address the resulting oxidative stress as a potential therapeutic option
Glutathione injection is used by doctors to detox the body of the poisonous side effects of medical treatments such as chemotherapy
A clear demonstration was obtained that BPC 157 stimulated both the egr-1 gene (critical in proliferation, differentiation, and inflammation during cholestatic liver injury, cytokine and growth factor generation, and early extracellular matrix (collagen) formation) (Kim et al., 2006
see figure 13 in [10]