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69 In gastric cancer cells, the absence of CDO1 contributed to restore cellular glutathione levels, to prevent ROS and lipid peroxidation, and to promote resistance to ferroptosis, a type of programmed cell death dependent on iron and associated with lipid peroxide accumulation

Inflammatory responses to monomeric and aggregated -Synuclein in peripheral blood of Parkinson disease patients

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The mechanism of ferroptosis and applications in tumor treatment