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The antiglycation potential of H1 receptor antagonists in vitro studies in bovine serum albumin model and in Silico molecular docking analyses

This microenvironment comprises abnormal blood and lymphatic vessels, extracellular matrix networks, cancer-associated fibroblasts, immune cells, metabolic gradients and soluble mediators, with substantial variation across tumor types, between patients and among regions within the same lesion

At the cellular level, thiol antioxidants induced apoptosis in transformed cell lines but not in normal cells, suggesting that direct redox control of p53 may be influential in determining cell fate under stress conditions [28]

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