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These SCFAs stimulate the secretion of GLP-1, improving metabolic function

however, catabolic processes, after all, result in starving cells to spare nutrients, hence sustaining cellular processes, and maintaining survival

A growing number of evidence indicates that GLP-1-based therapies have anti-inflammatory effects in various cells, tissues, and organs by reducing the synthesis of pro-inflammatory cytokines and limiting immune cell infiltration

And of course we have a number of patients with chronic kidney disease who, as you mentioned, pass away presumably prematurely in the absence of the availability of these drugs from cardiovascular causes

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