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The biotransformation that leads to the delayed formation of thiomethyl metabolites via the thiomethyl shunt pathway has been shown to occur in rodents via the enterohepatic circulation and biliary excretion of the glutathione-derived conjugates into the intestine ( 9 )

In studies examining muscle regeneration following injury, NNMT inhibition promoted stem cell activation and improved healing outcomes

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This reduced frequency can enhance patient adherence and convenience, potentially improving treatment outcomes