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The same review discusses its role in tissue protection, wound healing, vascular function, and inflammatory modulation

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This chapter the role of redox state in apoptosis perturbation, exploring how the imbalance in redox status not only can directly responsible for tissue damage, but also promote development through the imbalance of immunological tolerance and the generation of neo-antigenic determinants from one side and of autoantibodies from the other

(c) nuclear translocation of predominantly p65/p50 dimers to induce transcription of target genes, including well-established FLICE-like inhibitory protein (c-FLIP) and apoptosis protein inhibitors (c-IAP)

Cancer Res Serine Biosynthesis Is a Metabolic Vulnerability in IDH2-Driven Breast Cancer Progression