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Importantly, overexpression of the wild-type (WT) AMPK1 catalytic subunit in shGDH1 cells not only increased cytosolic NADPH level and alleviated disulfide stress (Fig

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As an inhibitor of NRF2, Keap1 recognizes and binds to the ETGE and DLG motifs of NRF2 via its Kelch repeat domain, thereby continuously suppressing and promoting the degradation of NRF2 [87]

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IGF-1 LR3 activates IGF1R with high affinity and can activate hybrid IGF1R/IR receptors to some degree

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