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Microglia and macrophage exhibit attenuated inflammatory response and ferroptosis resistance after RSL3 stimulation via increasing Nrf2 expression

The extrinsic apoptosis pathway is triggered by death receptor-death ligand interactions, including FasL/FasR, TRAIL/TRAIL-R1 or TRAIL-R2, and TNF/TNF-R1 [77,78,79]

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It is not a therapeutic good, is not registered on the ARTG, and has not been assessed by the TGA for safety, quality, or efficacy

Metabolic disturbances and endocrine dysregulation in diabetic states may inhibit CYP11A1 expression, reducing testosterone synthesis and further aggravating testicular dysfunction [152]

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