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Mediators Inflamm 2017:4810258
Beyond these established roles, excess lactate can directly influence gene expression and protein function through lysine lactylation, thereby establishing a mechanistic link between altered metabolism and sustained epigenetic regulation (Zhang et al., 2019
By selectively disrupting key antioxidant systemssuch as inhibiting glutathione synthesis with buthionine sulfoximine (BSO), blocking thioredoxin reductase with auranofin, or directly targeting NRF2therapies can push cancer cells beyond their limited oxidative buffering capacity
PubMed Link Lee DH, et al
Human clinical data for investigational peptides in bone healing is essentially nonexistent
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