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Additionally, targeting both c-Met and FGFR pathways provides superior suppression of HCC progression ( Recently, a growing body of evidence suggests that aberrant activation of HGF/c-Met signaling is associated with resistance of target therapies (Xiang et al., 2019) signaling pathways to induce the resistance to sorafenib

(v) 5 M antimycin A to inhibit complex III to prevent electron transfer to complex IV