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There are multiple compensatory mechanisms for glycocalyx damage, such as an increase in the expression of tight junction proteins and changes in cytoskeletal structure, which help maintain the normal function of microvessels (103) and further delay the effects

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M.St-GermainC.BlaisE.CrapoJ.et al (2016)

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High rates of liver cirrhosis and hepatocellular carcinoma in chronic hepatitis B patients with metabolic and cardiovascular comorbidities

At the network level, the end result is that the entrainment overrides pathological oscillatory activity