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Cardiac Repair Effects in Myocardial Infarction Models Documented and Research examining TB-500 in pre-clinical myocardial infarction models has documented significant cardiac repair effects

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S1PR2, located both in the liver and in the brain, activates the ERK and AKT pathways that regulate cell survival, inflammation, and stress response in the CNS

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A key aspect of the in vitro work was the meticulous quantification of GSH levels, as well as related markers such as glutathione disulfide, reactive oxygen species (ROS), and adenosine triphosphate (ATP)