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Therefore, we speculate that applying incretin-based medications in PD models can promote insulin secretion, improve peripheral and central insulin resistance, stabilize mitochondrial function, enhance energy metabolism, and exert neuroprotective effects that stabilize dopamine homeostasis (88)
methionine and tryptophan) or synthesized in too low concentrations (n-acetylcysteine (NAC)) by mammals and are therefore dietary essential nutrients [93]
This means the thyroid is, by design, continuously exposed to oxidative stress as a byproduct of its own hormone synthesis
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