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Abstract Purpose Currently, 31 patients with classical-like EDS (clEDS) due to tenascin-X deficiency have been reported in the literature

Y.SennettR.RezzaA.ClavelC.GrisantiL.ZemlaR.et al (2014)

Clinical Endocrinology (Oxford), 1986 Jun, Volume 24 (Issue 6), Pages 693-8

[92] Drug design and drug development exploits NAD + in three ways: as a direct target of drugs, by designing enzyme inhibitors or activators based on its structure that change the activity of NAD-dependent enzymes, and by trying to inhibit NAD + biosynthesis