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The authors also acknowledge the Department of Pharmaceuticals under the Ministry of Chemicals and Fertilizers, Government of India

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This redox imbalance promotes mitochondrial DNA damage, lipid peroxidation, and activation of apoptotic pathways, ultimately contributing to myofibrillar disorganisation and interstitial fibrosis (73)

A summary of the study characteristics and study quality can be found in Table 1 and Supplementary Material 3 respectively

Similarly, T4 has a short biological half-life, requiring frequent dosing to maintain therapeutic levels, which diminishes its clinical practicality (69)