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The clinical trial was 12 weeks, and it was powered as a proof-of-concept study

As mentioned previously, the A1 astrocyte phenotype, induced by microgliaderived IL1, TNF, and C1q, loses its neuroprotective functions and secretes neurotoxic and proinflammatory mediators that exacerbate neuronal damage

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fasting glucose at baseline and at 8 weeks The weekly DAC injection raises the logistical burden less but creates the tonic stimulation concern described above