On the one hand, the occurrence of MOMP will lead to the release of apoptosis-related proteins into the cytoplasm, thus causing the opening of the mitochondrial permeability transition pore, which in turn results in the disruption of mitochondrial respiration, matrix swelling, and rupture of the mitochondrial outer membrane, thereby inducing apoptosis
Glucagon-like peptide-1 (GLP-1) receptor agonists and neuroinflammation: implications for neurodegenerative disease treatment
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In addition to its role in energy production, ALC is a powerful antioxidant that helps protect neurons from the damaging effects of oxidative stress
329 Paradoxically, a study showed that AML cell death after APR-246 treatment is inhibited by iron chelators, inhibitors of lipid peroxidation, and lipophilic antioxidants and is characterized by the accumulation of lipid peroxidation, thus meeting the definition of ferroptosis
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