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The observation that FLNA 2124 which does not abolish the interaction of SSTR2 with endogenous FLNA, was able to block the apototic effects of SST2 and the SST2-mediated ERK1/2 inhibition suggests that the FLNA scaffold properties are required for the assembly of signal transduction complexes
Also supports angiogenesis for athletic recovery, cardiac health, and enhanced performance
Clinicians should be cautious and monitor for unexpected effects when combining BPC-157 with other treatments
This is the most important theoretical risk of this specific stack
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