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Mechanistically, we show that this effect occurs independently of canonical melatonin receptor signaling in ILC2s and instead requires activation of the NRF2-glutathione axis

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Elevated ACC glutamate has been directly associated with lack of remission in certain samples of chronic [13,14,15] or first-episode schizophrenia [16, 17] [UK sample], but this has not been a consistent observation

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