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doi: 10.1016/j.juro.2013.08.080 [DOI] [PubMed] [Google Scholar] 51.Crivelli JJ, Yan PL, Shahinian V, Hsi RS, Hollingsworth JM

The enhanced potency is due to the decreased binding of IGF1 LR3 to all known IGF binding proteins

Instead of injecting a synthetic version of the hormone from the outside, you reprogram cells to manufacture it internally

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When sprained or torn, these structures heal even more slowly than tendons due to even poorer blood supply

Tumor-associated macrophages (TAMs), important immunosuppressive cells in the microenvironment, are able to activate the Gli2/IGF-2/ERK1/2 signaling axis to promote TGF- secretion and thus mediate the migration, invasion and EMT of hepatocellular carcinoma cells (Huh-7 cells) (73)