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0.8% decrease

The phase 2 clinical trial data, published in The Lancet Diabetes and Endocrinology, tells a more nuanced story than the headlines suggest

These results indicate that the increased amount of non-12-OH FXR-antagonistic BAs consequent to the decrease in CYP8B1 expression after T3 treatment potentiates GLP-1 production by deactivating the intestinal FXR, thereby improving glucose homeostasis via the augmentation of insulin secretion

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