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Experimental vascular studies indicate that BPC-157 modulates endothelial nitric oxide synthase activity by altering the interaction between endothelial nitric oxide synthase and its regulatory binding partner caveolin-1 [1][2]

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Van Goethem G, Dermaut B, Lfgren A, Martin JJ, Van Broeckhoven C (2001) Mutation of POLG is associated with progressive external ophthalmoplegia characterized by mtDNA deletions

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