Thus, the transition from "physiological" to "pathological" fibrosis is driven by impaired apoptosis of myofibroblasts, as well as excessive ECM deposition and accumulation, resembling a persistent wound-healing response
3.6 Trimethylamine-N-oxide in non-alcoholic fatty liver disease A case-control and a cross-sectional study revealed that the presence and extremity of non-alcoholic fatty liver disease (NAFLD) were significantly correlated with higher circulating TMAO levels in Chinese adults (Zhang et al., 2021)
Teno JM, Mitchell SL, Skinner J, et al
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