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Research has shown that isoflavones and flavonoids of BP restrain the cell cycle at G1 and or G2/M stages and upregulate the expression of pro-apoptotic genes and dysfunction of the capability of enzymes associated with the growth and development of tumor cells while hindering the activity of intermediates such as fibroblast growth factor (FGF) and vascular endothelial growth factor (VEGF) and also inhibiting the production of ROS (177, 178)

IACS-010759, a selective mitochondrial complex I inhibitor, reduces tumorigenicity in acute myeloid leukemia models dependent on oxidative phosphorylation, with enhanced efficacy under glucose-restricted conditions

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Licht T et al (2016) VEGF preconditioning leads to stem cell remodeling and attenuates age-related decay of adult hippocampal neurogenesis

(2018) demonstrated that the individual diagnostic performance of HGF was inadequate