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While ongoing studies continue to explore its full range of mechanisms, BPC-157 remains an important compound for understanding how the body responds to injury and stress at the molecular level.

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Hajee Mohammad Danesh Science and Technology University, Bangladesh

It is currently in late-stage clinical trials (phase 3) and has not received a marketing authorisation from the Medicines and Healthcare products Regulatory Agency (MHRA) or the European Medicines Agency (EMA)

Thus, disruption of mitochondrial iron homeostasis may amplify oxidative injury to mitochondrial proteins, lipids, and DNA, compromise ATP generation, and ultimately trigger energy stress that converges on ferroptotic cell death

Endothelial protection: Prevents oxidative injury to vascular lining