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It also suggests that once mitochondrial injury has been initiated, its further progression may become increasingly independent of MCU itself owing to self-sustaining feed-forward mechanisms such as mPTP opening, oxidative stress, defective Ca 2+ extrusion, and inflammatory signaling

TL;DR There is no single answer

The proportions of acute exacerbation were 55% and 22% individually

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There are multiple metabolic factors that contribute to an individuals weight