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Further research using BNC1 / mice demonstrated that BNC1 defects promote intracellular iron accumulation and induce oocyte ferroptosis by activating the Merlin (NF2)/Hippo/YAP/TFRC/ACSL4 pathway (Wang F

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Therefore, depleting GSH to modulate ROS level holds significant promise as an effective therapeutic strategy for treating AML [9, 22, 23]

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doi: 10.1016/0277-9536(95)80007-7 185

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