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PAGln induces endothelial cell activation through increased reactive oxygen species leading to endothelial dysfunction To elucidate vascular endothelial dysfunction, we cultured HCAECs and exposed them to varying concentrations and durations of PAGln (Fig

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However, the remaining portion is converted by cytochrome P450 enzymes into N-acetyl-p-benzoquinone imine (NAPQI), a toxic metabolite