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Materials and methods The study was registered at ClinicalTrials.gov (id: NCT01312571), and approval was obtained from the regional ethics committee at Ume University, Sweden (dnr: 2015-310-31 M)

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It's described in the literature as the most selective reported to raise GH without meaningfully raising cortisol, prolactin, or appetite, unlike older GHRPs such as GHRP-6

By binding to p53, it induces nuclear exclusion, allowing p53 to translocate to the cytoplasm and mitochondria, subsequently activating the p53/BCL-2/Caspase-3 signaling pathway to execute selective apoptosis in senescent populations (Frontiers, 2025