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Further, the downstream effector molecules of mitochondrial-dependent apoptotic pathways were found to be activated such as levels of procaspase-3 41 were reduced suggesting its cleavage into active caspases (Fig

Eur J Endocrinol (2009) 160:74752.10.1530/EJE-09-0012 17 ColaoAAuriemmaRSLombardiGPivonelloR

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