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This pathway is critical for limiting the build up of harmful intermediates and maximizing protein turnover over short term [4143]

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This form forms a complex with CXCL12 to initiate leukocyte recruitment through CXCR4 interaction [49], while also activating the NF-B pathway through TLR2/4/9 to induce pro-inflammatory cytokine (IL-6, TNF-) secretion from monocytes

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