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The intracellular environment in macrophages has been strongly associated with the induction of metabolic indolence and antibiotic tolerance 7,8,9,10,11

This article explores the possible pathways by which intracellular enzymes convert cyanocobalamin, aquacobalamin (hydroxocobalamin), and methylcobalamin into active forms of vitamin B12

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In addition, TG2 expression is upregulated in many cancer cells 9,10,11,12