Meanwhile, excessive NO co-transmission can be induced by toxic forms of both amyloid and tau, which in turn both remodels neurotransmission and amplifies nitrogen stress in ways that can feed into AD pathology (Wang et al., 2023)
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Oti T, Sakamoto H
Despite multiple studies in the field, there is still no consensus on the most suitable animal models that could representatively demonstrate the pathogenesis of GC side effects
This makes the combination particularly well suited to mechanistic studies of receptor cross-talk, signal amplification, and pulse generation that single-receptor models cannot adequately replicate [2][3]
