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Other, rarer conditions affecting copper transport or metabolism also fall squarely into this category

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This is clearly demonstrated in people living with DS, where the overexpression of CBS is a genetic consequence that causes a significant reduction of serum homocysteine concentration leading to less available methionine within the methionine cycle

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GCLC, NQO1 and HO-1) and negative regulation on SREBP1 and the inflammatory response 48

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