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Interestingly, in a two year follow up study which was conducted 12 months after the subjects stopped receiving exenatide the researchers found that participants previously exposed to exenatide demonstrated a significant improvement (based on a blind assessment) in their motor features when compared to the control subjects involved in the study

2015, p

doi:10.1177/10600280251407145

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The pathways include inflammation, vascular damage, mitochondrial dysfunction, the effects of insulin resistance (IR) on metabolism in the brain, the formation of advanced glycation end products (AGEs), and the aggregation of pathological proteins such as A and -synuclein [12]

A glucagon-like peptide-1 analog reverses the molecular pathology and cardiac dysfunction of a mouse model of obesity