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The breadth of BPC-157s preclinical profile spanning gastrointestinal protection, musculoskeletal repair, angiogenesis, neuroprotection, and cytoprotection has sustained interest from research groups investigating tissue repair mechanisms and regenerative biology

The vitamin is then transferred to TC2, which serves as the circulating plasma transporter, hereditary defects in the production of TC2 and its receptor may produce functional deficiencies in B 12 and infantile megaloblastic anemia, and abnormal B 12 related biochemistry, even in some cases with normal blood B 12 levels

Naturally occurring plasma levels of GHK were confirmed to decline from approximately 200 g/L at age 2025 to 80 g/L by age 6080, coinciding with reduced regenerative capacity

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We also know that the serum B12 test is inaccurate and cannot tell us what is happening at a cellular level

Per-component dose ranges in published preclinical research differ across the two compounds