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IR induced metabolic disturbances lead to degradation of brain function, and prolonged uncontrolled high blood glucose levels adversely affect a variety of metabolic pathways such as oxidative stress, formation of advanced glycosylation end products (AGEs), and protein kinase C activation, while metabolic disturbances inducing cerebral microvascular damage affect the extent of cognitive impairment (1, 7)

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[3] The combination of the extension plus the E3R substitution is what distinguishes Long R3 IGF-1 from the simpler R3 IGF-1 analog and from native IGF-1

[PMC free article] [PubMed] [Google Scholar] 53.Effectiveness of N-acetylcysteine for preserving residual renal function in patients undergoing maintenance hemodialysis: multicenter randomized clinical trial