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This metabolic redundancy provides a mechanistic explanation for why CB-839, despite reducing cell viability, did not enhance 5-FU efficacy in our setting

Improvements strongly associated with the changes in the level of reduced glutathione and reduced/oxidized glutathione ratio

this is following previous reports on NTZ-ameliorative effects on liver injury in CCl4-induced hepatopathy and in steatohepatitis models (Walczak et al., 2018