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In double-blind studies, neither participants nor doctors know who receives the placebo or the active medication

cAMP sensor Epac as a determinant of ATP-sensitive potassium channel activity in human pancreatic cells and rat INS-1 cells

Based on these observations with GEP12, and on our unexpected findings that GEP44-mediated increases in ISR and decreases in glucagon release can be detected only in the presence of Y1-R antagonists, we proceeded to characterize the contributions of Y1-R to glucose uptake in muscle tissue